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If you’d like an appointment call Stan and Treva Voreyer

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Many of you know that I talk about triggerpoints as the most common source of pain in the body. I have found two website addresses that have all the trigger points and has a symptom index of common pains and ailments that may be caused by them. When you have a pain use the guide to help you find the muscle or muscles that may be causing it.

 

This is the page for upper body triggerpoints:

 

http://www.triggerpoints.net/_sgt/m1_1.htm

 

Lower body:

 

http://www.triggerpoints.net/_sgt/m2_1.htm

 

Related symptoms:

If you check here you’ll find it covers things like runny nose, Menstrual pain and lots more.

 

http://www.triggerpoints.net/_sgt/m3m2_1.htm

 

 

Its time for Fruits and Veggies here is a site that lists the residual pesticides in the most common fruit and veggies:

 

http://www.foodnews.org/

 

After you look through the list you should choose ORGANIC!!

 


NEWER information for women of all ages looking to potentially prevent osteoporosis


Osteoporosis prevention found in an often-overlooked hormone


By Jonathan V. Wright, M.D.

 

Historically, patent medicine companies have placed most of their “hormones against osteoporosis” bets on the estrogen side of the hormone equation. Makes sense. They’ve already got a patent “estrogen” called Premarin® that slows bone loss.
But there’s another hormone involved in bone health that’s at least as important as estrogen. It’s called progesterone. Studies have shown that declining progesterone production can cause bone loss to start in premenopausal women as young as 35 years old.

 

In fact, the decline in progesterone during the premenopausal years is associated with a decrease in bone mass of about 1 percent per year, which accelerates to about 3 percent to 5 percent during menopause.1

 

Your doctor likely won’t tell you about progesterone, though—at least not in relation to osteo- porosis—because conventional medicine has yet to come to grips with this particular benefit of the hormone. The mainstream primarily refers to this hormone for birth control pills and for HRT in menopausal women. But even in these circumstances, instead of using the bio-identical version of the hormone, doctors turn to the “extraterrestrial” (never before found on planet Earth), patent-medicine form: progestin.

 

Mainstream’s fight against natural hormones


Progesterone? Progestin? What’s the difference? Who cares, anyway? Mainstream physicians have learned to make no distinction between natural progesterone and synthetic progestin, which makes the people who sell patent medicines very happy. They’ve been going to a lot of trouble for years to blur the lines.


Patent medicine researchers, on the other hand, understand all too well the potential value of progesterone in maintaining healthy bones, but, because they can’t patent it (it doesn’t even require a prescription), they’ve gone off after bigger game—patentable progesterone look-alikes that might approach, but can never, ever, perfectly match progesterone’s unique, unpatentable molecular “fingerprint” in the body.


While it’s true that the synthetic progestins may actually have some bone-building benefits, taking them is certainly not worth the risks involved—especially since you can take the natural form, which has even more benefits. If you want a stark example of how progestins differ from bio-identical progesterone, consider these basic facts:

 

1) Progesterone has long been used to facilitate pregnancies (its name derives from its pro-gestational function), but Provera® and other progestins are known to cause birth defects. (Provera is the best known, most widely prescribed, and most dangerous of the progestins.)

 

2) The body easily converts progesterone into other important steroid hormones, including cortisol, aldosterone, and others, while “extraterrestrial” progestins, alien to human physiology, cannot be converted to other human steroids.2

 

Skeptics cite the lack of clinical evidence supporting progesterone’s role in bone metabolism, and it’s true—there aren’t any large, long-term, double-blind, placebo- controlled studies evaluating progesterone in people who are at risk of developing osteoporosis. Such studies are simply too expensive for anyone other than patent medicine companies to sponsor, and none of them are interested in researching a non-patentable hormone.

 

The publication deck is also stacked against progesterone. Medical journals, most of which depend on patent medicine company advertising to stay afloat, primarily publish progestin studies, the vast majority of which are sponsored directly or indirectly by—guess who?—patent medicine companies.

 

By contrast, published progesterone studies tend to be older, fewer, smaller, and often less well-controlled. And since most of the studies are from other countries (where English is not the primary language), it’s easier for conventional medicine to ignore or dismiss them. To further the dilemma, progesterone doesn’t have a multimillion dollar advertising and public relations structure, as the progestins do, to bring it to the attention of doctors and patients alike.

 

Of course, none of this discounts the powerful effect progesterone has on the body. A considerable body of evidence (from laboratory and small studies in actual menopausal women, and from years of close clinical observation by doctors who prescribe bio-identical hormone replacement therapy) all point to the same conclusion: Progesterone may be just as important as estrogen for bone health—and for dozens of other normal functions throughout a woman’s lifetime. (See the sidebar on page 3.)

 

The case for progesterone


For years, the prime mover behind many of the most important findings regarding progesterone in women has been the internationally known Canadian investigator, Jerilynn C. Prior, M.D., a Professor of Endocrinology and Metabolism at the University of British Columbia, Vancouver, BC, and Scientific Director of the Centre for Menstrual Cycle and Ovulation Research (www.cemcor.ubc.ca).


Dr. Prior elected to approach the progesterone question indirectly, by using small studies and focusing on the relationship between bone mineral density and natural hormone metabolism. Unlike research on patented “extraterrestrial molecule” progestins and osteoporosis drugs, she did not try to fool, alter, or overcome the natural processes.

 

Before I explain her basic hypothesis, it’s important to understand that progesterone has two major glands of origin. In both men and women progesterone is made in the adrenal glands. But women also produce progesterone in their ovaries.
Prior to ovulation, levels of progesterone in women are not greatly different from progesterone levels in men. But after ovulation, there is normally a tremendous surge in progesterone output by the ovaries that doesn’t subside until shortly before the menstrual period. Depending on the timing of ovulation, a healthy adult woman’s body has a much higher level of progesterone for as much as two weeks of every four-week cycle for 30 to 40 years. The potential decline in progesterone that can affect women’s bone health accounts for almost all of the progesterone that originates in the ovaries.


Based on this information, this was Dr. Prior’s basic hypothesis: If bone mineral density (BMD), a key index of bone strength, is directly related to progesterone levels, then bone mineral density should rise a little bit during each luteal phase (the 10 to 16 days after ovulation, when progesterone production is at its peak). Conversely, during the early days of the menstrual cycle, when progesterone levels are at their lowest, BMD should decline a little—even though estrogen levels are high.

 

The longer the luteal phase, the more progesterone is produced each month, and the denser the bones should be. If there is no ovulation during a particular cycle, progesterone is not produced, and for that cycle, BMD should decline.


To test this hypothesis, Dr. Prior measured vertebral BMD3 in 66 healthy premenopausal women at the start and again at the end of a 12-month period. The results showed that, over the year of the study, far and away the best predictor of changes in BMD was NOT the women’s estrogen levels, diet, exercise, or body shape or size––as the mainstream would have you believe—but rather the length of their luteal phases.4

 

In another study, Dr. Prior measured vertebral BMD in a group of 66 young premenopausal women (from 21 to 42 years old), who had shortened luteal phases and/or an occasional anovulatory cycle (no ovulation, no luteal phase), and, consequently, no progesterone release. The results showed that these deviations from the normal hormonal balance were strongly associated with declining BMD, accounting for 24 percent of the lost bone.

 

The women who maintained normal menstrual cycles and who had the highest levels of progesterone gained spinal BMD at a rate of about 1 percent per year. By contrast, the women with shortened cycles lost 2.8 percent of their BMD, and those who had anovulatory cycles (and consequently had the lowest progesterone levels of all) lost 4.2 percent of their BMD per year—more than double the average bone loss seen in all the women in that study.5

 

In a third study, Dr. Prior followed a similar group of premenopausal women (with an average age of about 41 years old) for five years. Despite their continuing ovulation —and essentially normal estrogen levels with no hints of impending menopause—the women nevertheless lost 6 percent of their vertebral bone mass over the five-year period. Again, the best and earliest predictor of bone loss was their luteal phase length during the first year of the study.6

 

Overall, Dr. Prior’s findings lead us to a startling conclusion: Bone loss actually begins long before menopause—sometimes as early as age 30—while estrogen levels are still essentially normal, but progesterone production has already begun its decline. A woman can lose as much as 75 percent of her progesterone between the ages of 35 and 50. Thus, it’s quite common for women to start menopause with only 25 percent of their youthful progesterone on board.

 

These studies reveal just how sensitive your BMD is to even small, normal, periodic shifts in progesterone level. Remember, the women in these studies were all premenopausal and still menstruating on a regular basis. Yet, even small variations in their apparent progesterone levels could significantly reduce the growth of new bone tissue.


The importance of replacing progesterone before menopause


Now, it’s one thing to argue that declining progesterone levels contribute to postmenopausal bone loss because it places a damper on new bone building. But, it’s something altogether different to claim that progesterone replacement can prevent —or even reverse—osteoporosis.

 

While the available evidence for the former is quite strong, the evidence for the latter isn’t quite as definitive. Nevertheless, the benefits of progesterone to healthy bones are obvious — especially to those physicians who have been prescribing bio-identical hormone replacement therapy (BHRT). So if I were you, I wouldn’t wait around for the double-blind, placebo-controlled studies to come trickling in. Who knows how much your bone density will have dwindled by then.

 

One clue that your progesterone may be lower than optimal is the absence of a reliable, every-month mid-cycle temperature rise, checkable very simply with a thermometer. (If you don’t know how to do this, check with your physician.) Another clue might be hearing the phrase: “You’re having PMS, dear.”


Instead, if you’re a woman over 35 (or younger, in some cases), please have your progesterone levels checked by a physician skilled and knowledgeable in both natural therapies and bio-identical hormone replacement. If you find your progesterone levels are lower than the usual post-ovulatory levels (perhaps you’re not ovulating), it may be possible to restore ovulation and/or higher progesterone levels without taking any bio-identical progesterone from the outside.

 

If that doesn’t work for you, your physician can help you to find the right amount (not too little, not too much) and timing for bio-identical progesterone replacement, even if menopause is years away. Your bones and the rest of your body will be healthier for it!


*This article adapted from the upcoming 2nd edition (2007) of Bio-identical Hormone Replacement for Women Over 45 by Lane Lenard Ph.D. and Jonathan V. Wright, first edition 1997.